A UC San Francisco study of human and mouse pancreatic tissue suggests a new origin story for type 1 (T1) diabetes. The findings flip current assumptions about the causes of the disease on their head and demonstrate a promising new preventative strategy that dramatically reduced disease risk in laboratory animals.
T1 diabetes is an autoimmune disorder that usually begins in childhood or adolescence, and has generally been attributed to the body’s immune system mistakenly attacking the insulin-producing beta cells of the pancreas. Scientists still don’t understand what triggers this autoimmune response, and so far, attempts to develop therapies to protect or restore beta cells have not been successful.
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